Please use this identifier to cite or link to this item: https://doi.org/10.3389/fimmu.2016.00463
Title: Malaria parasites: The great escape
Authors: Rénia, L 
Goh, Y.S
Keywords: epitope
gamma interferon
glycosylphosphatidylinositol
heat shock protein 70
immunoglobulin G1
immunoglobulin G3
immunoglobulin M
intercellular adhesion molecule 1
vaccine
antigenic variation
B lymphocyte
CD4+ T lymphocyte
cell adhesion
cell invasion
complement system
cross reaction
histology
immune deficiency
immune evasion
Kupffer cell
malaria falciparum
nonhuman
parasite survival
phagocytosis
Plasmodium
protein polymorphism
Review
Issue Date: 2016
Citation: Rénia, L, Goh, Y.S (2016). Malaria parasites: The great escape. Frontiers in Immunology 7 (NOV) : 463. ScholarBank@NUS Repository. https://doi.org/10.3389/fimmu.2016.00463
Rights: Attribution 4.0 International
Abstract: Parasites of the genus Plasmodium have a complex life cycle. They alternate between their final mosquito host and their intermediate hosts. The parasite can be either extra- or intracellular, depending on the stage of development. By modifying their shape, motility, and metabolic requirements, the parasite adapts to the different environments in their different hosts. The parasite has evolved to escape the multiple immune mechanisms in the host that try to block parasite development at the different stages of their development. In this article, we describe the mechanisms reported thus far that allow the Plasmodium parasite to evade innate and adaptive immune responses. © 2016 Rénia and Goh.
Source Title: Frontiers in Immunology
URI: https://scholarbank.nus.edu.sg/handle/10635/179902
ISSN: 16643224
DOI: 10.3389/fimmu.2016.00463
Rights: Attribution 4.0 International
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