Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML

Please use this identifier to cite or link to this item: https://doi.org/10.1182/blood-2008-05-156422

Title:	Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML
Authors:	Zhou, J. Bi, C. Janakakumara, J.V. Liu, S.-C. Chng, W.-J. Tay, K.-G. Poon, L.-F. Xie, Z. Palaniyandi, S. Chen, C.-S. Yu, H. Glaser, K.B. Albert, D.H. Davidsen, S.K.
Issue Date:	2009
Citation:	Zhou, J., Bi, C., Janakakumara, J.V., Liu, S.-C., Chng, W.-J., Tay, K.-G., Poon, L.-F., Xie, Z., Palaniyandi, S., Chen, C.-S., Yu, H., Glaser, K.B., Albert, D.H., Davidsen, S.K. (2009). Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML. Blood 113 (17) : 4052-4062. ScholarBank@NUS Repository. https://doi.org/10.1182/blood-2008-05-156422
Source Title:	Blood
URI:	http://scholarbank.nus.edu.sg/handle/10635/26991
ISSN:	00064971 15280020
DOI:	10.1182/blood-2008-05-156422
Appears in Collections:	Staff Publications

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