Please use this identifier to cite or link to this item:
https://doi.org/10.1111/j.1440-169X.2009.01163.x
DC Field | Value | |
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dc.title | Alternative splicing of SMADs in differentiation and tissue homeostasis | |
dc.contributor.author | Tao, S. | |
dc.contributor.author | Sampath, K. | |
dc.date.accessioned | 2014-10-27T08:47:46Z | |
dc.date.available | 2014-10-27T08:47:46Z | |
dc.date.issued | 2010-05 | |
dc.identifier.citation | Tao, S., Sampath, K. (2010-05). Alternative splicing of SMADs in differentiation and tissue homeostasis. Development Growth and Differentiation 52 (4) : 335-342. ScholarBank@NUS Repository. https://doi.org/10.1111/j.1440-169X.2009.01163.x | |
dc.identifier.issn | 00121592 | |
dc.identifier.uri | http://scholarbank.nus.edu.sg/handle/10635/102401 | |
dc.description.abstract | Tissue-specific alternate splicing is an important means of regulating gene expression during development. The effector proteins for the transforming growth factor-β signaling pathway, the SMADs, encode distinct isoforms generated via alternate splicing, which appear to have distinct tissue-specific expression profiles and functions. Here, we discuss the roles of various SMAD isoforms, and the consequences of mis-regulation of SMAD splicing in development and tissue homeostasis. © 2010 Japanese Society of Developmental Biologists. | |
dc.description.uri | http://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1111/j.1440-169X.2009.01163.x | |
dc.source | Scopus | |
dc.subject | RNA splicing | |
dc.subject | SMADs | |
dc.subject | TGF-β/SMAD signalling | |
dc.subject | Tissue homeostasis | |
dc.type | Review | |
dc.contributor.department | BIOLOGICAL SCIENCES | |
dc.description.doi | 10.1111/j.1440-169X.2009.01163.x | |
dc.description.sourcetitle | Development Growth and Differentiation | |
dc.description.volume | 52 | |
dc.description.issue | 4 | |
dc.description.page | 335-342 | |
dc.description.coden | DGDFA | |
dc.identifier.isiut | 000277084500001 | |
Appears in Collections: | Staff Publications |
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