Full Name
Peter Edward Lobie
(not current staff)
Variants
Lobie, P.E.
 
 
 
Email
phcpel@nus.edu.sg
 
Other emails
 

Publications

Results 61-76 of 76 (Search time: 0.011 seconds).

Issue DateTitleAuthor(s)
6126-Jan-2013Prognostic significance of the expression of GFRα1, GFRα3 and Syndecan-3, Proteins binding ARTEMIN, In mammary carcinomaWu, Z.-S.; Pandey, V.; Wu, W.-Y.; Ye, S.; Zhu, T.; Lobie, P.E. 
21-Jan-2021Putting the BRK on breast cancer: From molecular target to therapeuticsAng, Hui Li; Yuan, Yi ; Lai, Xianning ; Tan, Tuan Zea ; Wang, Lingzhi ; Huang, Benjamin BoJun; Pandey, Vijay; Huang, Ruby Yun-Ju; Lobie, Peter E ; Goh, Boon Cher ; Sethi, Gautam ; Yap, Celestial T ; Chan, Ching Wan ; Lee, Soo Chin ; Kumar, Alan Prem 
32017Release of HER2 repression of trefoil factor 3 (TFF3) expression mediates trastuzumab resistance in HER2+/ER+ mammary carcinomaChong, Q.-Y ; You, M.-L ; Pandey, V ; Banerjee, A ; Chen, Y.-J; Poh, H.-M; Zhang, M; Ma, L; Zhu, T; Basappa, Laboratory of Chemical Biology, Department of Chemistry, Bangalore University, Central College Campus, Bangalore, India; Liu, L; Lobie, P.E 
41-Dec-2013SHON is a novel estrogen-regulated oncogene in mammary carcinoma that predicts patient response to endocrine therapyJung, Y.; Abdel-Fatah, T.M.A.; Chan, S.Y.T.; Nolan, C.C.; Green, A.R.; Ellis, I.O.; Li, L.; Huang, B.; Lu, J.; Xu, B.; Chen, L.; Ma, R.Z.; Zhang, M.; Wang, J.; Wu, Z.; Zhu, T.; Perry, J.K.; Lobie, P.E. ; Liu, D.-X.
518-Jul-2003Src-CrkII-C3G-dependent activation of Rap1 switches growth hormone-stimulated p44/42 MAP kinase and JNK/SAPK activitiesLing, L.; Zhu, T.; Lobie, P.E. 
6Sep-2010STAT3α is oncogenic for endometrial carcinoma cells and mediates the oncogenic effects of autocrine human growth hormoneTang, J.-Z.; Kong, X.-J.; Banerjee, A.; Muniraj, N.; Pandey, V.; Steiner, M.; Perry, J.K.; Zhu, T.; Liu, D.-X.; Lobie, P.E. 
72018The GDNF Family: a Role in Cancer?Fielder, G.C.; Yang, T.W.-S.; Razdan, M.; Li, Y.; Lu, J.; Perry, J.K.; Lobie, P.E. ; Liu, D.-X.
14821-Feb-2003The growth hormone-binding protein is a location-dependent cytokine receptor transcriptional enhancerGraichen, R.; Sandstedt, J.; Goh, E.L.K.; Isaksson, O.G.P.; Törnell, J.; Lobie, P.E. 
14921-Feb-2012The HOXB7 protein renders breast cancer cells resistant to tamoxifen through activation of the EGFR pathwayJin, K.; Kong, X.; Shah, T.; Penet, M.-F.; Wildes, F.; Sgroi, D.C.; Ma, X.-J.; Huang, Y.; Kallioniemi, A.; Landberg, G.; Bieche, I.; Wu, X.; Lobie, P.E. ; Davidson, N.E.; Bhujwalla, Z.M.; Zhu, T.; Sukumar, S.
150Dec-2012The prolactin receptor mediates HOXA1-stimulated oncogenicity in mammary carcinoma cellsHou, L.; Xu, B.; Mohankumar, K.M.; Goffin, V.; Perry, J.K.; Lobie, P.E. ; Liu, D.-X.
15110-May-2013Trefoil factor 1 suppression of E-CADHERIN enhances prostate carcinoma cell invasiveness and metastasisBougen, N.M.; Amiry, N.; Yuan, Y.; Kong, X.J.; Pandey, V.; Vidal, L.J.P.; Perry, J.K.; Zhu, T.; Lobie, P.E. 
12Dec-2010Trefoil factor 3 is oncogenic and mediates anti-estrogen resistance in human mammary carcinomaKannan, N.; Kang, I.; Kong, X.; Tang, J.; Perry, J.K.; Mohankumar, K.M.; Miller, L.D.; Liu, E.T.; Mertani, H.C.; Zhu, T.; Grandison, P.M.; Liu, D.-X.; Lobie, P.E. 
132017Trefoil factor 3 mediation of oncogenicity and chemoresistance in hepatocellular carcinoma is AKT-BCL-2 dependentYou, M.-L ; Chen, Y.-J ; Chong, Q.-Y ; Wu, M.-M; Pandey, V ; Chen, R.-M ; Liu, L; Ma, L; Wu, Z.-S; Zhu, T; Lobie, P.E 
142014Trefoil factor 3 promotes metastatic seeding and predicts poor survival outcome of patients with mammary carcinomaPandey, V ; Wu, Z.-S; Zhang, M; Li, R; Zhang, J; Zhu, T; Lobie, P.E 
152015Trefoil factor-3 (TFF3) stimulates de novo angiogenesis in mammary carcinoma both directly and indirectly via IL-8/CXCR2Lau W.-H.; Pandey V.; Kong X.; Wang X.-N.; Wu Z.; Zhu T.; Lobie P.E. 
16Oct-2011Tumor expression of human growth hormone and human prolactin predict a worse survival outcome in patients with mammary or endometrial carcinomaWu, Z.-S.; Yang, K.; Wan, Y.; Qian, P.-X.; Perry, J.K.; Chiesa, J.; Mertani, H.C.; Zhu, T.; Lobie, P.E.