Please use this identifier to cite or link to this item: https://doi.org/10.1016/j.bbrc.2006.11.038
DC FieldValue
dc.titlePromotion of atherogenesis by copper or iron-Which is more likely?
dc.contributor.authorRajendran, R.
dc.contributor.authorRen, M.
dc.contributor.authorWatt, F.
dc.contributor.authorNing, P.
dc.contributor.authorHalliwell, B.
dc.contributor.authorTan, Kwong Huat B.
dc.date.accessioned2011-11-29T06:10:44Z
dc.date.available2011-11-29T06:10:44Z
dc.date.issued2007
dc.identifier.citationRajendran, R., Ren, M., Watt, F., Ning, P., Halliwell, B., Tan, Kwong Huat B. (2007). Promotion of atherogenesis by copper or iron-Which is more likely?. Biochemical and Biophysical Research Communications 353 (1) : 6-10. ScholarBank@NUS Repository. https://doi.org/10.1016/j.bbrc.2006.11.038
dc.identifier.issn0006291X
dc.identifier.issn10902104
dc.identifier.urihttp://scholarbank.nus.edu.sg/handle/10635/28897
dc.description.abstractIron levels increase in atherosclerotic lesions in cholesterol fed-rabbits and play a role in atherosclerosis. We investigated whether copper also rises. Male New Zealand White rabbits were fed high-cholesterol diets for 8 weeks. After sacrifice, lesion sizes were determined, and elemental analyses of the lesion and unaffected artery wall performed using nuclear microscopy. Unlike iron, lesion copper is decreased by about half compared with the unaffected artery wall, and much less copper than iron is present. Our data suggest that iron may be more likely to play a role in the promotion of atherosclerosis than copper. © 2006 Elsevier Inc. All rights reserved.
dc.description.urihttp://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1016/j.bbrc.2006.11.038
dc.sourceScopus
dc.subjectCopper
dc.subjectElemental analysis
dc.subjectIron
dc.subjectNuclear microscopy
dc.typeArticle
dc.contributor.departmentBIOCHEMISTRY
dc.contributor.departmentPHYSICS
dc.contributor.departmentPHARMACOLOGY
dc.description.doi10.1016/j.bbrc.2006.11.038
dc.description.sourcetitleBiochemical and Biophysical Research Communications
dc.description.volume353
dc.description.issue1
dc.description.page6-10
dc.description.codenBBRCA
dc.identifier.isiut000243336900002
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