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Please use this identifier to cite or link to this item: https://scholarbank.nus.edu.sg/handle/10635/25105
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dc.titleEnhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML
dc.contributor.authorZhou, J.
dc.contributor.authorBi, C.
dc.contributor.authorJanakakumara, J.V.
dc.contributor.authorLiu, S.-C.
dc.contributor.authorChng, W.-J.
dc.contributor.authorTay, K.-G.
dc.contributor.authorPoon, L.-F.
dc.contributor.authorXie, Z.
dc.contributor.authorPalaniyandi, S.
dc.contributor.authorChen, C.-S.
dc.contributor.authorYu, H.
dc.contributor.authorGlaser, K.B.
dc.contributor.authorAlbert, D.H.
dc.contributor.authorDavidsen, S.K.
dc.date.accessioned2011-08-01T03:04:55Z
dc.date.available2011-08-01T03:04:55Z
dc.date.issued2009
dc.identifier.citationZhou, J., Bi, C., Janakakumara, J.V., Liu, S.-C., Chng, W.-J., Tay, K.-G., Poon, L.-F., Xie, Z., Palaniyandi, S., Chen, C.-S., Yu, H., Glaser, K.B., Albert, D.H., Davidsen, S.K. (2009). Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML. Blood 113 (17) : 4052-4062. ScholarBank@NUS Repository.
dc.identifier.issn00064971
dc.identifier.issn15280020
dc.identifier.urihttp://scholarbank.nus.edu.sg/handle/10635/25105
dc.description.urihttp://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1182/blood-2008-05-156422
dc.sourceScopus
dc.typeArticle
dc.contributor.departmentPHYSIOLOGY
dc.contributor.departmentMEDICINE
dc.contributor.departmentCANCER SCIENCE INSTITUTE OF SINGAPORE
dc.description.sourcetitleBlood
dc.description.volume113
dc.description.issue17
dc.description.page4052-4062
dc.identifier.isiut000265447000025
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