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Please use this identifier to cite or link to this item: https://doi.org/10.1074/jbc.M113.536300
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dc.titleThe expression of Fn14 via mechanical stress-activated JNK contributes to apoptosis induction in osteoblasts
dc.contributor.authorMatsui, H.
dc.contributor.authorFukuno, N.
dc.contributor.authorKanda, Y.
dc.contributor.authorKantoh, Y.
dc.contributor.authorChida, T.
dc.contributor.authorNagaura, Y.
dc.contributor.authorSuzuki, O.
dc.contributor.authorNishitoh, H.
dc.contributor.authorTakeda, K.
dc.contributor.authorIchijo, H.
dc.contributor.authorSawada, Y.
dc.contributor.authorSasaki, K.
dc.contributor.authorKobayashi, T.
dc.contributor.authorTamura, S.
dc.date.accessioned2016-09-06T05:44:01Z
dc.date.available2016-09-06T05:44:01Z
dc.date.issued2014-03-07
dc.identifier.citationMatsui, H., Fukuno, N., Kanda, Y., Kantoh, Y., Chida, T., Nagaura, Y., Suzuki, O., Nishitoh, H., Takeda, K., Ichijo, H., Sawada, Y., Sasaki, K., Kobayashi, T., Tamura, S. (2014-03-07). The expression of Fn14 via mechanical stress-activated JNK contributes to apoptosis induction in osteoblasts. Journal of Biological Chemistry 289 (10) : 6438-6450. ScholarBank@NUS Repository. https://doi.org/10.1074/jbc.M113.536300
dc.identifier.issn1083351X
dc.identifier.urihttp://scholarbank.nus.edu.sg/handle/10635/126623
dc.description.abstractBackground: Fn14 is a highly inducible member of the TNF receptor family. Results: Large-magnitude mechanical stress induced Fn14 expression via JNK in osteoblasts. Conclusion: Expression of Fn14 regulates mechanical stress-induced apoptosis in osteoblasts. Significance: This is the first elucidation of the mechanism of excessive mechanical stress-induced apoptosis mediated by Fn14. © 2014 by The American Society for Biochemistry and Molecular Biology, Inc.
dc.description.urihttp://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1074/jbc.M113.536300
dc.sourceScopus
dc.typeArticle
dc.contributor.departmentBIOLOGICAL SCIENCES
dc.description.doi10.1074/jbc.M113.536300
dc.description.sourcetitleJournal of Biological Chemistry
dc.description.volume289
dc.description.issue10
dc.description.page6438-6450
dc.description.codenJBCHA
dc.identifier.isiut000332389400012
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