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Please use this identifier to cite or link to this item: https://doi.org/10.1016/j.cmet.2010.09.007
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dc.titleAn IRKO in the Podo: Impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy
dc.contributor.authorGurley, S.B.
dc.contributor.authorCoffman, T.M.
dc.date.accessioned2016-06-01T10:31:42Z
dc.date.available2016-06-01T10:31:42Z
dc.date.issued2010-10-06
dc.identifier.citationGurley, S.B., Coffman, T.M. (2010-10-06). An IRKO in the Podo: Impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy. Cell Metabolism 12 (4) : 311-312. ScholarBank@NUS Repository. https://doi.org/10.1016/j.cmet.2010.09.007
dc.identifier.issn15504131
dc.identifier.urihttp://scholarbank.nus.edu.sg/handle/10635/124848
dc.description.abstractDespite its high prevalence, the mechanisms causing diabetic kidney disease remain poorly understood. In this issue of Cell Metabolism, Welsh et al. (2010) show that elimination of insulin receptors from the glomerular podocyte, a cell that is central to the pathogenesis of proteinuric renal diseases, recapitulates many features of human diabetic nephropathy. © 2010 Elsevier Inc.
dc.description.urihttp://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1016/j.cmet.2010.09.007
dc.sourceScopus
dc.typeOthers
dc.contributor.departmentDUKE-NUS GRADUATE MEDICAL SCHOOL S'PORE
dc.description.doi10.1016/j.cmet.2010.09.007
dc.description.sourcetitleCell Metabolism
dc.description.volume12
dc.description.issue4
dc.description.page311-312
dc.identifier.isiut000282836800004
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