An IRKO in the Podo: Impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy

Please use this identifier to cite or link to this item: https://doi.org/10.1016/j.cmet.2010.09.007

Title:	An IRKO in the Podo: Impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy
Authors:	Gurley, S.B. Coffman, T.M.
Issue Date:	6-Oct-2010
Citation:	Gurley, S.B., Coffman, T.M. (2010-10-06). An IRKO in the Podo: Impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy. Cell Metabolism 12 (4) : 311-312. ScholarBank@NUS Repository. https://doi.org/10.1016/j.cmet.2010.09.007
Abstract:	Despite its high prevalence, the mechanisms causing diabetic kidney disease remain poorly understood. In this issue of Cell Metabolism, Welsh et al. (2010) show that elimination of insulin receptors from the glomerular podocyte, a cell that is central to the pathogenesis of proteinuric renal diseases, recapitulates many features of human diabetic nephropathy. © 2010 Elsevier Inc.
Source Title:	Cell Metabolism
URI:	http://scholarbank.nus.edu.sg/handle/10635/124848
ISSN:	15504131
DOI:	10.1016/j.cmet.2010.09.007
Appears in Collections:	Staff Publications

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