Please use this identifier to cite or link to this item: https://doi.org/10.1016/j.tibs.2006.07.005
DC FieldValue
dc.titlePhagocyte-derived reactive species: salvation or suicide?
dc.contributor.authorHalliwell, B.
dc.date.accessioned2011-11-29T05:59:09Z
dc.date.available2011-11-29T05:59:09Z
dc.date.issued2006
dc.identifier.citationHalliwell, B. (2006). Phagocyte-derived reactive species: salvation or suicide?. Trends in Biochemical Sciences 31 (9) : 509-515. ScholarBank@NUS Repository. https://doi.org/10.1016/j.tibs.2006.07.005
dc.identifier.issn09680004
dc.identifier.urihttp://scholarbank.nus.edu.sg/handle/10635/28763
dc.description.abstractActivated phagocytes produce 'reactive oxygen, halogen and nitrogen species' that help to kill some types of microorganism. How these species destroy microorganisms remains, however, an enigma: both direct oxidative damage and indirect damage (whereby reactive species promote the actions of other antibacterial agents) are involved, and no single mechanism is likely to account for the killing of all microorganisms. Phagocyte-derived reactive species are known to injure human tissues and to contribute to inflammation. Recently, however, we have learned that they can also be anti-inflammatory by modulating the immune response. These data have implications for the proposed use of antioxidants to treat inflammation. © 2006 Elsevier Ltd. All rights reserved.
dc.description.urihttp://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1016/j.tibs.2006.07.005
dc.sourceScopus
dc.typeReview
dc.contributor.departmentBIOCHEMISTRY
dc.description.doi10.1016/j.tibs.2006.07.005
dc.description.sourcetitleTrends in Biochemical Sciences
dc.description.volume31
dc.description.issue9
dc.description.page509-515
dc.description.codenTBSCD
dc.identifier.isiut000240789600006
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