Please use this identifier to cite or link to this item: https://doi.org/10.1016/0140-6736(91)91710-C
DC FieldValue
dc.titleHyperinsulinaemia in thyrotoxic hypokalaemic periodic paralysis
dc.contributor.authorLee, K.-O.
dc.contributor.authorTaylor, E.A.
dc.contributor.authorOh, V.M.
dc.contributor.authorCheah, J.-S.
dc.contributor.authorAw, S.-E.
dc.date.accessioned2016-11-28T10:17:22Z
dc.date.available2016-11-28T10:17:22Z
dc.date.issued1991
dc.identifier.citationLee, K.-O., Taylor, E.A., Oh, V.M., Cheah, J.-S., Aw, S.-E. (1991). Hyperinsulinaemia in thyrotoxic hypokalaemic periodic paralysis. Lancet 337 (8749) : 1063-1064. ScholarBank@NUS Repository. https://doi.org/10.1016/0140-6736(91)91710-C
dc.identifier.issn01406736
dc.identifier.urihttp://scholarbank.nus.edu.sg/handle/10635/131199
dc.description.abstractTo test the hypothesis that there is an abnormal serum insulin response to a carbohydrate load in thyrotoxic hypokalaemic periodic paralysis (THPP), 18 men with THPP and 15 with uncomplicated thyrotoxicosis were studied during an oral glucose tolerance test. The THPP group had significantly higher fasting insulin concentrations (27.6 [3.6] vs 13.4 [1.8] mU/l; p < 0.005) and a higher overall insulin response to oral glucose (p < 0.001 by ANOVA) than the thyrotoxicosis group. There were no significant differences in fasting or stimulated glucose. Hyperinsulinaemia may be an important factor in the precipitation of acute paralysis in THPP.
dc.description.urihttp://libproxy1.nus.edu.sg/login?url=http://dx.doi.org/10.1016/0140-6736(91)91710-C
dc.sourceScopus
dc.typeArticle
dc.contributor.departmentMEDICINE
dc.description.doi10.1016/0140-6736(91)91710-C
dc.description.sourcetitleLancet
dc.description.volume337
dc.description.issue8749
dc.description.page1063-1064
dc.description.codenLANCA
dc.identifier.isiutA1991FK20400007
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