1. ScholarBank@NUS

HUANG QING

rp05005

Collaboration Map View Statistics Email Alert RSS Feed

Profile

Full Name
HUANG QING
(not current staff)
Variants
Huang, Q.
 
Main Affiliation
 
Faculty
 
Email
ephhq@nus.edu.sg
 

Publications

Results 1-19 of 19 (Search time: 0.004 seconds).

Issue DateTitleAuthor(s)
12009Activation of the PI3K-Akt-mTOR signaling pathway promotes necrotic cell death via suppression of autophagyWu, Y.-T.; Tan, H.-L.; Huang, Q. ; Ong, C.-N. ; Shen, H.-M. 
216-Jan-2013AMPK mediates a pro-survival autophagy downstream of PARP-1 activation in response to DNA alkylating agentsZhou, J. ; Ng, S.; Huang, Q. ; Wu, Y.-T.; Li, Z.; Yao, S.Q. ; Shen, H.-M. 
32007Anti-cancer properties of anthraquinones from rhubarbHuang, Q. ; Lu, G. ; Shen, H.-M.; Choon, N.O.; Chung, M.C.M. 
42008Autophagy plays a protective role during zVAD-induced necrotic cell deathWu, Y.-T.; Tan, H.-L.; Huang, Q. ; Ong, C.-N.; Shen, H.-M.; Kim, Y.-S.; Liu, Z.-G.; Pan, N.; Ong, W.-Y.; Shen, H.-M.
52010Chrysin sensitizes tumor necrosis factor-α-induced apoptosis in human tumor cells via suppression of nuclear factor-kappaBLi, X.; Yang, X.-F.; Huang, Q. ; Ong, C.-N. ; Shen, H.-M. 
62010Dual role of 3-methyladenine in modulation of autophagy via different temporal patterns of inhibition on class I and III phosphoinositide 3-kinaseWu, Y.-T.; Tan, H.-L.; Huang, Q. ; Ong, C.-N. ; Shen, H.-M. ; Shui, G. ; Wenk, M.R. ; Bauvy, C.; Codogno, P.
72010Dual role of 3-methyladenine in modulation of autophagy via different temporal patterns of inhibition on class I and III phosphoinositide 3-kinaseWu, Y.-T.; Tan, H.-L.; Huang, Q. ; Ong, C.-N. ; Shen, H.-M. ; Shui, G. ; Wenk, M.R. ; Bauvy, C.; Codogno, P.
82006Emodin inhibits tumor cell adhesion through disruption of the membrane lipid raft-associated integrin signaling pathwayHuang, Q. ; Shen, H.-M. ; Ong, C.-N. ; Shui, G. ; Wenk, M.R. 
92006Emodin inhibits tumor cell adhesion through disruption of the membrane lipid raft-associated integrin signaling pathwayHuang, Q. ; Shen, H.-M.; Ong, C.-N.; Shui, G. ; Wenk, M.R. 
10May-2005Emodin inhibits tumor cell migration through suppression of the phosphatidylinositol 3-kinase-Cdc42/Rac1 pathwayHuang, Q. ; Shen, H.-M. ; Ong, C.-N. 
1116-Nov-2010Erratum to: Huang Q, Shen H-M. To die or to live: The dual role of poly(ADP-ribose) polymerase-1 in autophagy and necrosis under oxidative stress and DNA damage (Autophagy (2009) 5, (273-276))Huang, Q. ; Shen, H.-M. 
122007Hypericin photoactivation triggers down-regulation of matrix metalloproteinase-9 expression in well-differentiated human nasopharyngeal cancer cellsDu, H.-Y.; Bay, B.-H. ; Du, H.-Y.; Olivo, M.; Mahendran, R. ; Huang, Q. ; Shen, H.-M.; Ong, C.-N.
132007Hypericin photoactivation triggers down-regulation of matrix metalloproteinase-9 expression in well-differentiated human nasopharyngeal cancer cellsDu, H.-Y.; Bay, B.-H. ; Olivo, M.; Mahendran, R. ; Huang, Q. ; Shen, H.-M. ; Ong, C.-N. 
142007Hypericin photoactivation triggers down-regulation of matrix metalloproteinase-9 expression in well-differentiated human nasopharyngeal cancer cellsDu, H.-Y.; Bay, B.-H. ; Olivo, M. ; Mahendran, R. ; Huang, Q. ; Shen, H.-M. ; Ong, C.-N. 
1515-Jul-2004Inhibitory effect of emodin on tumor invasion through suppression of activator protein-1 and nuclear factor-κBHuang, Q. ; Shen, H.-M. ; Ong, C.-N. 
162007Luteolin sensitizes the anticancer effect of cisplatin via c-Jun NH2-terminal kinase-mediated p53 phosphorylation and stabilizationShi, R.; Huang, Q. ; Ong, Y.-B.; Ong, C.-N.; Shen, H.-M.; Zhu, X.; Zhao, B.; Lu, J.
172006Methyl-3-indolylacetate inhibits cancer cell invasion by targeting MEK1/2-ERK1/2 signaling pathwayZhang, S. ; Huang, Q. ; Shen, H.-M.; Ong, C.-N.; Li, Z.; Wu, X.
182009To die or to live: The dual role of poly(ADP-ribose) polymerase-1 in autophagy and necrosis under oxidative stress and DNA damageHuang, Q. ; Shen, H.-M.
19Jan-2011ZVAD-induced necroptosis in L929 cells depends on autocrine production of TNFα mediated by the PKC-MAPKs-AP-1 pathwayWu, Y.-T.; Tan, H.-L.; Huang, Q. ; Sun, X.-J.; Zhu, X.; Shen, H.-M.