1. ScholarBank@NUS

Kaur,Charanjit

rp04486

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Full Name
Kaur,Charanjit
(not current staff)
Variants
Kaur C.
Kaur, C.
 
Main Affiliation
 
Faculty
 
Email
antkaurc@nus.edu.sg
 

Publications

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Policy:  Open
Author:  Dheen, S Thameem

Results 1-8 of 8 (Search time: 0.008 seconds).

Issue DateTitleAuthor(s)
11-Jan-2003Distribution of NADPH-diaphorase and expression of nNOS, N-methyl-D-aspartate receptor (NMDAR1) and non-NMDA glutamate receptor (GlutR2) genes in the neurons of the hippocampus after domoic acid-induced lesions in adult ratsAnanth, C; Dheen, ST ; Gopalakrishnakone, P ; Kaur, C 
22011Expression of sphingosine kinase 1 in amoeboid microglial cells in the corpus callosum of postnatal ratsLin H.; Baby N. ; Lu J.; Kaur C. ; Zhang C.; Xu J.; Ling E.-A. ; Dheen S.T. 
31-Aug-2007From blood to brain: amoeboid microglial cell, a nascent macrophage and its functions in developing brainKaur, Charanjit ; Dheen, S Thameem ; Ling, Eng-ang 
425-Jul-2018Hypoxia-induced histone modifications in activated microglia in vitroKrishnan, Jayabharathi ; Karthikeyan, Aparna ; Kaur, Charanjit ; Thameem Dheen S 
52013Notch-1 Signaling Regulates Microglia Activation via NF-κB Pathway after Hypoxic Exposure In Vivo and In VitroYao L.; Kan E.M.; Kaur C. ; Dheen S.T. ; Hao A.; Lu J.; Ling E.-A. 
61-Sep-2010Nuclear Factor-kappa B/p65 Responds to Changes in the Notch Signaling Pathway in Murine BV-2 Cells and in Amoeboid Microglia in Postnatal Rats Treated With the gamma-Secretase Complex Blocker DAPTCao, Q; Li, P; Lu, J; DHEEN, ST ; Kaur, C ; Ling, E-A 
719-Oct-2017Production of proinflammatory mediators in activated microglia is synergistically regulated by Notch-1, glycogen synthase kinase (GSK-3 beta) and NF-kappa B/p65 signallingCao, Qiong; Karthikeyan, Aparna ; Dheen, S Thameem ; Kaur, Charanjit ; Ling, Eng-Ang 
82013Toll-like receptor 4 mediates microglial activation and production of inflammatory mediators in neonatal rat brain following hypoxia: Role of TLR4 in hypoxic microgliaYao L.; Kan E.M.; Lu J.; Hao A.; Dheen S.T. ; Kaur C. ; Ling E.-A.