Skip navigation
IVLE
Email
Library
Map
Calendar
Home
Research Outputs
View research outputs
Deposit publication / dataset
Researchers
Help
FAQs
Contact us
Guidelines
Log in
ScholarBank@NUS
Research Outputs
Browsing by Author
Chong Khin Soon Terence
Enter a last name
Select a letter below to browse by last name or type
0-9
A
B
C
D
E
F
G
H
I
J
K
L
M
N
O
P
Q
R
S
T
U
V
W
X
Y
Z
Or
Results/Page
5
10
15
20
25
30
35
40
45
50
55
60
65
70
75
80
85
90
95
100
Showing results 1 to 7 of 7
Refman
EndNote
Bibtex
RefWorks
Excel
CSV
PDF
Send via email
Issue Date
Title
Author(s)
Aug-1993
Nuclear factor I and epithelial cell-specific transcription of human papillomavirus type 16
Apt, D.
;
Chong, T.
;
Liu, Y.
;
Bernard, H.-U.
1989
Numerous nuclear proteins bind the long control region of human papillomavirus type 16: A subset of 6 of 23 DNase I-protected segments coincides with the location of the cell-type-specific enhancer
Gloss, B.
;
Chong, T.
;
Bernard, H.-U.
1991
Sequence variants of human papillomavirus type 16 in clinical samples permit verification and extension of epidemiological studies and construction of a phylogenetic tree
Ho, L.
;
Chan, S.-Y.
;
Chow, V.
;
Chong, T.
;
Tay, S.-K.
;
Villa, L.L.
;
Bernard, H.-U.
1995
Suppression of the yeast mutation rft1-1 by human p53
Koerte, A.
;
Chong, T.
;
Li, X.
;
Wahane, K.
;
Cai, M.
Nov-1991
The enhancer of human papillomavirus type 16: Binding sites for the ubiquitous transcription factors oct-1, NFA, TEF-2, NF1, and AP-1 participate in epithelial cell-specific transcription
Chong, T.
;
Apt, D.
;
Gloss, B.
;
Isa, M.
;
Bernard, H.-U.
25-Feb-1990
Transcription of the transforming genes of the oncogenic human papillomavirus-16 is stimulated by tumor promotors through AP1 binding sites
Chan, W.-K.
;
Chong, T.
;
Bernard, H.-U.
;
Klock, G.
11-Feb-1990
Transcriptional activation of human papillomavirus 16 by nuclear factor I, AP1, steroid receptors and a possibly novel transcription factor, PVF: A model for the composition of genital papillomavirus enhancers
Chong, T.
;
Chan, W.-K.
;
Bernard, H.-U.